I have written about the mechanism that causes potential cancer due to COVID vaccination over the past four years.
And, of course, the key to protecting the body from cancer is the immune system. What we do not want is that it becomes dysfunction and dysfunction.
Immune adjustment failure
The immune system is highly adjusted by the interconnected route that the immunistics are still discovered, and affects another part by changing a certain part. If SARS-COV-2 vaccination is changing something in the immune system, via changes in vaccine MRNA code, it will adversely affect receptors like Toll or other means. Can you change the change?
Rin, etc. Please explain the importance of the receptor like a traffic fee Polimar-TLR-7/8 Agonist (Ajubant) for CD8 T cell induction and mechanism: 'Chop molecular fee receptor -7 and -8agonist (TLR-7/8A) can be used as vaccine Ajuvant. Induces CD8 T cell immunity. (Emphasized has been added)
Invivogen, summary craft et al. (2005) and Reece et al. (2005), We will support the discovery: 'TLR7-8 Agonist's ability to activate DC Therefore, it triggers TH1 and CD8+ T cells. You can exploit the response to enhance the effectiveness of vaccination. (Emphasized has been added)
There are important connections from those authors with Toll -sama receptor, dendritic cells (DC), T cells, especially CD8 T cells. Toll -like receptor activates dendritic cells and triggers reactions from CD8 T cells. CD8 T cells are established as an important part of the immune system Not only defense against infection, but also. FU and Jiang, in Cardiic cells and CD8 T cell immunity in the tumor microscopic environmentWe will further explore complex interactions and clarify the importance of dendritic cells in the battle with CD8 T cells. “The direct presentation of the tumor antigen by tumor cells is an important role in the Effector function of the CD8 T cells, and the premise of professional antigen that presents intersection -cells. In particular, DC is required to maintain the CD8 T cells and cell poisonous immunity response. (Emphasis has been added).
As the author states, the receptors, such as the toll, are needed to activate dendritic cells, and when dendritic cells maintain T cell cell -poisonous killing to cancer tumors. What happens if the Toll -like receptor is not working so well? Increased cancer? And infection?
Receptors 7 and 8 like traffic fees The literature explains that it is important in bringing out important CD8 T cell response. With that in mind, let's remember what Dr. Weissman and Dr. Carico wrote. Suppression of RNA recognition by toll -like receptor: Effect of nucleoside modifications and evolution of RNA (2005): 'RNA indicates that it incorporates the incorporation of the modified nucleoside M5C, M6A, M5U, S2U, or pseudo -urzin, although it is signal through human TLR3, TLR7, and TLR8.
There is a technology that exists Used in SARS-COV-2 vaccineTurn off the TLR 7 and 8 signals The immune system needs to fight infection and cancer.
In addition, repeated COVID vaccination, UVERSKY et al. ExplainedIt can lead to an increase in IgG4 antibodies with negative results.
However, new evidence suggests that the IgG4 -level report detected after repeated vaccination with the MRNA vaccine may not be a protective mechanism. Rather, suppressing natural antiviral reactions configure immune-resistant mechanisms for spike proteins that can promote SARS-COV2 infection and duplication without opposition. Because of the high antigen concentration, MRNA vaccination has been repeated, so the increase in IGG4 synthesis may cause autoimmune disease. Promotes the growth of cancer Self -immunity myocarditis in individuals who are easily affected. (Emphasized has been added)
Latest research
The latest research to raise concerns has just been published and has been entitled. Proteomics and mastrophy evaluation of MRNA-1273 and BNT162B2 vaccine in human recipe serum. There are some points to consider in this study. Their analysis showed the up regulation of glutathione -specific gamma -glutamyl sichlotransferase 1 (CHAC1). The prosperous glutathione enzyme CHAC1 acts on glutathione, and glutathione plays a major role in health. I explained the role of Kennedy:
Glutathion (GSH) is the most abundant non -protein -sular that exists in the mammol concentration of mammal tissue. It functions as an important intracellular antioxidant as a cell dodox state that protects cells from damage caused by lipid peroxide, reactive oxygen, nitrogen species, and biological foreign substances. Recent studies emphasize the importance of GSH in major signal reactions as controllers of cell differentiation, proliferation, apoptosis, ferroptosis, and immune function. The molecular changes in the GSH antioxidant system and the obstacle of GSH normal gender are related to the start, progress, and therapeutic reaction of the tumor.
Also, consider the role of CHAC 1 and Glutathione. Developed protein response。 If the UPR is not functioning properly, it could lead to a fatal pathology, including incorrectly folded proteins, such as dementia.
(CHAC1 catalyst) Glutathione 5-oxo L-Proline and Cys-Gly dipeptide. It acts especially on glutathion, but does not work on other gamma -glutamil peptides ()PUBMED: 27913623) The depletion of glutathion is an important factor in the start and execution of apoptosis. By intervening the apoptosis promotion effect of ATF4-ATF3-DDIT3/CHOP cascade, it functions as an apoptosis promotion component of the developed protein response route ().PUBMED: 19109178)
Author Proteomics and serum evaluation of reactions to MRNA-1273 and BNT162B2 vaccine in human recipe sera Hickey et al. Also, note:
The excessive expression of enzymes causes glutathione depletion, which adversely affects the balance of cells between active oxygen species and antioxidant defense.
Therefore, it is important to balance glutathiones, and an unusual up regulation of enzymes that affects how much glutathion is available may show a serious problem. Too much less can lead to fatal pathology, including cancer.
Hickey et al. They also demonstrated the up regulation of cancer/testicular antigen 1 (CTAG1A | CTAG1B) in the serum of the BNT162B2 recipient, and they pointed out.
Cancer -Testis antigen is an antigen identified with various malignant tumors and is usually expressed only in testicular tissue.
In light of the previous research results and the latest serum results, there is a very high concern that these vaccines can contribute to serious and fatal diseases like cancer or cause fatal diseases. It is.
Of course, we wait for the latest serum research to confirm that it can be confirmed, but the risks of these vaccines are still on the risk side.
